Runx2 alleviates high glucose-suppressed osteogenic differentiation via PI3K/AKT/GSK3β/β-catenin pathway-Reference-Cited by-同舟云学术

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Runx2 alleviates high glucose-suppressed osteogenic differentiation via PI3K/AKT/GSK3β/β-catenin pathway

Published:2017-05-18 Issue:8 Volume:41 Page:822-832
ISSN:1065-6995
Container-title:Cell Biology International
language:en
Short-container-title:Cell Biol Int

Author:

Chen Yang¹²³,Hu Yun¹²³,Yang Lan¹²³,Zhou Jie¹²³,Tang Yuying¹²³,Zheng Leilei¹²³,Qin Pu¹²³

Affiliation:

1. College of Stomatology; Chongqing Medical University; Chongqing 401147 China

2. Chongqing Key Laboratory of Oral Diseases and Biomedical Sciences; Chongqing 401147 China

3. Chongqing Municipal Key Laboratory of Oral Biomedical Engineering of Higher Education; Chongqing 401147 China

Funder

National Natural Science Foundation of China

Natural Science Foundation of Chongqing

Program for the Medical Scientific Research Project of Chongqing

Publisher

Wiley

Subject

Cell Biology,General Medicine

Reference32 articles.

1. Chronic hyperglycemia modulates osteoblast gene expression through osmotic and non-osmotic pathways;Botolin;J Cell Biochem,2006

2. Role and regulation of RUNX2 in osteogenesis;Bruderer;Eur Cell Mater,2014

3. Akt interacts directly with Smad3 to regulate the sensitivity to TGF-beta induced apoptosis;Conery;Nat Cell Biol,2004

4. Wnt induction of chondrocyte hypertrophy through the Runx2 transcription factor;Dong;J Cell Physiol,2006

5. Phosphorylation of beta-catenin by AKT promotes beta-catenin transcriptional activity;Fang;J Biol Chem,2007

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