Pathogenesis of diquat-induced liver necrosis in selenium-deficient rats: Assessment of the roles of lipid peroxidation and selenoprotein P
Author:
Publisher
Wiley
Subject
Hepatology
Reference34 articles.
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4. Non-cyclooxygenase-derived prostanoids (F2-isoprostanes) are formed in situ on phospholipids.
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1. Selenoproteins Protect Against Avian Liver Necrosis by Metabolizing Peroxides and Regulating Receptor Interacting Serine Threonine Kinase 1/Receptor Interacting Serine Threonine Kinase 3/Mixed Lineage Kinase Domain-Like and Mitogen-Activated Protein Kinase Signaling;Frontiers in Physiology;2021-08-12
2. Dietary glucose oxidase and/or catalase supplementation alleviates intestinal oxidative stress induced by diquat in weaned piglets;Animal Science Journal;2021-01
3. Novel tumour suppressor roles for GZMA and RASGRP1 in Theileria annulata ‐transformed macrophages and human B lymphoma cells;Cellular Microbiology;2020-09-29
4. Selenium-Enriched Yeast Alleviates Oxidative Stress-Induced Intestinal Mucosa Disruption in Weaned Pigs;Oxidative Medicine and Cellular Longevity;2020-03-19
5. F 2 ‐Isoprostanes as Markers of Oxidant Stress: An Overview;Current Protocols in Toxicology;2005-05
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