PTPRGinhibition by DNA methylation and cooperation withRASgene activation in childhood acute lymphoblastic leukemia-Reference-Cited by-同舟云学术

PTPRGinhibition by DNA methylation and cooperation withRASgene activation in childhood acute lymphoblastic leukemia

Published:2014-02-19 Issue:5 Volume:135 Page:1101-1109
ISSN:0020-7136
Container-title:International Journal of Cancer
language:en
Short-container-title:Int. J. Cancer

Author:

Xiao Jianqiao¹,Lee Seung-Tae¹²,Xiao Yuanyuan¹,Ma Xiaomei³,Andres Houseman E.⁴,Hsu Ling-I⁵,Roy Ritu⁶,Wrensch Margaret⁷,de Smith Adam J.¹,Chokkalingam Anand⁵,Buffler Patricia⁵,Wiencke John K.⁷,Wiemels Joseph L.¹

Affiliation:

1. Department of Epidemiology and Biostatistics; University of California San Francisco; San Francisco CA

2. Department of Laboratory Medicine and Genetics; Samsung Medical Center, Sungkyunkwan University School of Medicine; Seoul Korea

3. Department of Epidemiology and Public Health; Yale University; New Haven CT

4. College of Public Health and Human Sciences; Oregon State University; Corvallis OR

5. Division of Epidemiology; School of Public Health, University of California Berkeley; Berkeley CA

6. Cancer Center Institute, University of California San Francisco; San Francisco CA

7. Department of Neurological Surgery; University of California San Francisco; San Francisco CA

Funder

National Institutes of Health

Tobacco-Related Disease Research Program

Leukemia and Lymphoma Society

Publisher

Wiley

Subject

Cancer Research,Oncology

Reference33 articles.

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2. Backtracking RAS mutations in high hyperdiploid childhood acute lymphoblastic leukemia;Wiemels;Blood Cells Mol Dis,2010

3. Somatic activation of oncogenic KRAS in hematopoietic cells initiates a rapidly fatal myeloproliferative disorder;Braun;Proc Natl Acad Sci U S A,2004

4. Response and resistance to MEK inhibition in leukaemias initiated by hyperactive Ras;Lauchle;Nature,2009

5. Key pathways are frequently mutated in high-risk childhood acute lymphoblastic leukemia: a report from the Children's Oncology Group;Zhang;Blood,2011

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