Role of Fas and Treg Cells in Fracture Healing as Characterized in the Fas‐Deficient (lpr) Mouse Model of Lupus-Reference-Cited by-同舟云学术

Role of Fas and Treg Cells in Fracture Healing as Characterized in the Fas‐Deficient (lpr) Mouse Model of Lupus

Published:2014-05-19 Issue:6 Volume:29 Page:1478-1491
ISSN:0884-0431
Container-title:Journal of Bone and Mineral Research
language:en
Short-container-title:J Bone Miner Res

Author:

Al‐Sebaei Maisa O¹²,Daukss Dana M¹,Belkina Anna C³,Kakar Sanjeev¹,Wigner Nathan A¹,Cusher Daniel¹,Graves Dana⁴,Einhorn Thomas¹,Morgan Elise¹⁵,Gerstenfeld Louis C¹³

Affiliation:

1. Orthopaedic Research LaboratoryBoston University School of MedicineBostonMAUSA

2. King Abdul Aziz University, Faculty of DentistryDepartment of Oral and Maxillofacial SurgeryJeddahSaudi Arabia

3. Flow Cytometry Core FacilityBoston University School of MedicineBostonMAUSA

4. University of Pennsylvania School of Dental MedicinePhiladelphiaPAUSA

5. Department of Biomedical EngineeringBoston University School of EngineeringBostonMAUSA

Publisher

Wiley

Subject

Orthopedics and Sports Medicine,Endocrinology, Diabetes and Metabolism

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jbmr.2169

Reference57 articles.

1. Expression of Osteoprotegerin, Receptor Activator of NF‐κB Ligand (Osteoprotegerin Ligand) and Related Proinflammatory Cytokines During Fracture Healing

2. Impaired Fracture Healing in the Absence of TNF-α Signaling: The Role of TNF-α in Endochondral Cartilage Resorption

3. Tumor necrosis factor alpha (TNF-α) coordinately regulates the expression of specific matrix metalloproteinases (MMPS) and angiogenic factors during fracture healing

4. Impaired Intramembranous Bone Formation during Bone Repair in the Absence of Tumor Necrosis Factor-Alpha Signaling

5. TNF- promotes fracture repair by augmenting the recruitment and differentiation of muscle-derived stromal cells

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