Enriched <scp>PUFA</scp> environment of <i>Leishmania infantum</i> promastigotes promotes the accumulation of lipid mediators and favors parasite infectivity towards <scp>J774</scp> murine macrophages-Reference-Cited by-同舟云学术

Enriched PUFA environment of Leishmania infantum promastigotes promotes the accumulation of lipid mediators and favors parasite infectivity towards J774 murine macrophages

Published:2022-12-21 Issue:2 Volume:58 Page:81-92
ISSN:0024-4201
Container-title:Lipids
language:en
Short-container-title:Lipids

Author:

Leroux Marine¹,Bouazizi‐Ben Messaoud Hana¹,Luquain‐Costaz Céline¹²,Jordheim Lars P.³,Le Faouder Pauline⁴,Gustin Marie‐Paule⁵,Aoun Karim⁶,Lawton Philippe¹,Azzouz‐Maache Samira¹,Delton Isabelle¹²^ORCID

Affiliation:

1. CNRS 5007, LAGEPP University of Lyon, Université Claude Bernard Lyon 1 Villeurbanne France

2. INSA‐Lyon, Department of Biosciences Villeurbanne France

3. Inserm 1052, CNRS 5286, Centre Léon Bérard, Centre de Recherche en Cancérologie de Lyon University of Lyon, Université Claude Bernard Lyon 1 Lyon France

4. MetaToul‐Lipidomic Core Facility MetaboHUB, I2MC Inserm U1048 Toulouse France

5. Inserm 1111, CNRS UMR5308, Centre International de Recherche en Infectiologie, ENS de Lyon University of Lyon, Université Claude Bernard Lyon 1 Lyon France

6. Pasteur Institute of Tunis, LR 11‐IPT‐06 Laboratory of Medical Parasitology, Biotechnology and Biomolecules University Tunis El Manar Tunis Tunisia

Funder

INSA de Rouen

UCBL

Publisher

Wiley

Subject

Cell Biology,Organic Chemistry,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/lipd.12365

Reference37 articles.

1. Lipid and fatty acid metabolism in trypanosomatids

2. Prostaglandin E2/Leukotriene B4 balance induced by Lutzomyia longipalpis saliva favors Leishmania infantum infection

3. Uptake, Distribution, and Oxidation of Fatty Acids by Leishmania mexicana Amastigotes

4. A RAPID METHOD OF TOTAL LIPID EXTRACTION AND PURIFICATION

5. Lipids From Trypanosoma cruzi Amastigotes of RA and K98 Strains Generate a Pro-inflammatory Response via TLR2/6

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