Glutamate decarboxylase inhibition and vitamin B6metabolism in brain of cirrhotic rats chronically treated with carbon tetrachloride
Author:
Publisher
Wiley
Subject
Cellular and Molecular Neuroscience
Reference28 articles.
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3. Biochemistry and physiology of brain ammonia;Cooper;Physiol Rev,1987
4. Regional brain GABA metabolism and release during hepatic coma produced in rats chronically treated with carbon tetrachloride;Díaz-Muñoz;Neurochem Res,1988
5. Determination of serum proteins by means of the biuret reaction;Garnall;J Biol Chem,1949
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1. RATS MADE CIRRHOTIC BY CHRONIC CCl4TREATMENT STILL EXHIBIT ANTICIPATORY ACTIVITY TO A RESTRICTED FEEDING SCHEDULE;Chronobiology International;2002-01
2. Effect of carbon tetrachloride on inositol 1,4,5-trisphosphate dependent and independent regulation of rat brain microsomal Ca2+ flux;Cellular Signalling;1994-07
3. Convulsions and inhibition of glutamate decarboxylase by pyridoxal phosphate-?-glutamyl hydrazone in the developing rat;Neurochemical Research;1994-02
4. Glutamate decarboxylase activity in the substantia nigra and the hippocampus of rats microinjected with inhibitors of the enzyme;Neurochemical Research;1991-03
5. Functional changes of brain mitochondria during experimental hepatic encephalopathy;Biochemical Pharmacology;1989-11
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