Prospective study of body size throughout the life-course and the incidence of endometrial cancer among premenopausal and postmenopausal women

Author:

Dougan Marcelle M.1,Hankinson Susan E.1234,Vivo Immaculata De345,Tworoger Shelley S.134,Glynn Robert J.67,Michels Karin B.1348

Affiliation:

1. Department of Epidemiology; Harvard School of Public Health; Boston MA

2. Division of Biostatistics and Epidemiology; University of Massachusetts School of Public Health and Health Sciences; Amherst MA

3. Channing Division of Network Medicine; Brigham and Women's Hospital, Harvard Medical School; Boston MA

4. Department of Medicine; Brigham and Women's Hospital, Harvard Medical School; Boston MA

5. Department of Epidemiology; Program in Genetic Epidemiology and Statistical Genetics, Harvard School of Public Health; Boston MA

6. Center for Cardiovascular Disease Prevention; Division of Preventive Medicine; Brigham and Women's Hospital, Harvard Medical School; Boston MA

7. Department of Biostatistics; Harvard School of Public Health; Boston MA

8. Obstetrics and Gynecology Epidemiology Center; Department of Obstetrics; Gynecology and Reproductive Biology; Brigham and Women's Hospital, Harvard Medical School; Boston MA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Cancer Research,Oncology

Reference50 articles.

1. National Cancer Institute http://www.cancer.gov/cancertopics/types/endometrial 2014

2. Overweight as an avoidable cause of cancer in Europe;Bergström;Int J Cancer,2001

3. Obesity, endogenous hormones and endometrial cancer risk: a synthetic review;Kaaks;Cancer Epidemiol Biomarkers Prev,2002

4. Estrogen induces endometrial cancer cell proliferation and invasion by regulating the fat mass and obesity-associated gene via PI3K/AKT and MAPK signaling pathways;Zhang;Cancer Lett,2012

5. Circulating adipokine levels and endometrial cancer risk in the prostate, lung, colorectal, and ovarian cancer screening trial;Luhn;Cancer Epidemiol Biomarkers Prev,2013

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