MicroRNA‐210 repression facilitates advanced glycation end‐product (AGE)‐induced cardiac mitochondrial dysfunction and apoptosis via JNK activation

Author:

Lin Kuan‐Ho12,Ng Shang‐Chuan34,Paul Catherine R.5,Chen Hong‐Chen67,Zeng Ren‐You34,Liu Jian‐Sheng38,Padma Viswanadha V.9,Huang Chih‐Yang510111213ORCID,Kuo Wei‐Wen34ORCID

Affiliation:

1. College of Medicine China Medical University Taichung Taiwan ROC

2. Department of Emergency Medicine China Medical University Hospital Taichung Taiwan ROC

3. Department of Biological Science and Technology, College of Life Sciences China Medical University Taichung Taiwan ROC

4. PhD Program for Biotechnology Industry China Medical University Taichung Taiwan ROC

5. Center of General Education, Buddhist Tzu Chi Medical Foundation Tzu Chi University of Science and Technology Hualien Taiwan ROC

6. Institute of Biochemistry and Molecular Biology National Yang‐Ming University Taipei Taiwan ROC

7. Cancer Progression Research Center National Yang‐Ming University Taipei Taiwan ROC

8. Department of Thoracic China Medical University Beigang Hospital Yunlin Taiwan ROC

9. Cardiovascular and Mitochondrial Related Disease Research Center, Hualien Tzu Chi Hospital Buddhist Tzu Chi Medical Foundation Hualien Taiwan ROC

10. Department of Biotechnology, Translational Research Laboratory, School of Biotechnology and Genetic Engineering Bharathiar University Coimbatore Tamil Nadu India

11. Graduate Institute of Biomedical Sciences China Medical University Taichung Taiwan ROC

12. Department of Medical Laboratory Science and Biotechnology Asia University Taichung Taiwan ROC

13. Department of Medical Research, China Medical University Hospital China Medical University Taichung Taiwan ROC

Funder

Ministry of Science and Technology, Taiwan

China Medical University Hospital

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

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