Sickle Cell Disease as a Multifactorial Condition
Author:
Publisher
Wiley
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/9780470015902.a0006021.pub2
Reference60 articles.
1. The influence of uridine diphosphate glucuronosyl transferase 1A promoter polymorphisms, betaS-globin gene haplotype, co-inherited alpha-thalassemia trait and Hb F on steady-state serum bilirubin levels in sickle cell anemia
2. Association of Polymorphisms ofIGF1Rand Genes in the Transforming Growth Factor–β/Bone Morphogenetic Protein Pathway with Bacteremia in Sickle Cell Anemia
3. Identification of genetic polymorphisms associated with risk for pulmonary hypertension in sickle cell disease
4. Association of klotho, bone morphogenic protein 6, and annexin A2 polymorphisms with sickle cell osteonecrosis
5. G6PD deficiency, absence of α-thalassemia, and hemolytic rate at baseline are significant independent risk factors for abnormally high cerebral velocities in patients with sickle cell anemia
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