Non‐ B DNA Structure and Mutations Causing Human Genetic Disease
Author:
Publisher
Wiley
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/9780470015902.a0022657.pub2
Reference60 articles.
1. Non-B DNA Conformations, Genomic Rearrangements, and Human Disease
2. Translocation and deletion breakpoints in cancer genomes are associated with potential non-B DNA-forming sequences
3. FANCJ is essential to maintain microsatellite structure genome-wide during replication stress
4. A recurrent 14q32.2 microdeletion mediated by expanded TGG repeats
5. A familial inverted duplication/deletion of 2p25.1–25.3 provides new clues on the genesis of inverted duplications
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