Haploinsufficiency of NADPH Oxidase Subunit Neutrophil Cytosolic Factor 2 Is Sufficient to Accelerate Full-Blown Lupus in NZM 2328 Mice
Author:
Affiliation:
1. University of Southern California School of Medicine; Los Angeles
2. Washington University School of Medicine in St. Louis; St. Louis Missouri
3. National Institute of Arthritis and Musculoskeletal and Skin Diseases, NIH; Bethesda Maryland
Funder
National Institutes of Health
Alliance for Lupus Research
Children's Discovery Institute at Washington University and St. Louis Children's Hospital
Publisher
Wiley
Subject
Immunology,Rheumatology,Immunology and Allergy
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/art.40141/fullpdf
Reference44 articles.
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2. Lupus-associated causal mutation in neutrophil cytosolic factor 2 (NCF2) brings unique insights to the structure and function of NADPH oxidase;Jacob;Proc Natl Acad Sci U S A,2012
3. Allelic heterogeneity in NCF2 associated with systemic lupus erythematosus (SLE) susceptibility across four ethnic populations;Kim-Howard;Hum Mol Genet,2014
4. Systemic lupus erythematosus-associated neutrophil cytosolic factor 2 mutation affects the structure of NADPH oxidase complex;Armstrong;J Biol Chem,2015
5. Regulation of the NADPH oxidase and associated ion fluxes during phagocytosis;Nunes;Traffic,2013
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