Overactivation of the TGF-β pathway confers a mesenchymal-like phenotype and CXCR4-dependent migratory properties to liver tumor cells-Reference-Cited by-同舟云学术

Overactivation of the TGF-β pathway confers a mesenchymal-like phenotype and CXCR4-dependent migratory properties to liver tumor cells

Published:2013-10-11 Issue:6 Volume:58 Page:2032-2044
ISSN:0270-9139
Container-title:Hepatology
language:en
Short-container-title:Hepatology

Author:

Bertran Esther¹,Crosas-Molist Eva¹,Sancho Patricia¹,Caja Laia¹,Lopez-Luque Judit¹,Navarro Estanislao¹,Egea Gustavo²,Lastra Raquel³,Serrano Teresa⁴,Ramos Emilio³,Fabregat Isabel¹⁵

Affiliation:

1. Bellvitge Biomedical Research Institute (IDIBELL); L'Hospitalet de Llobregat; Barcelona Spain

2. Department of Cell Biology, Immunology and Neuroscience; University of Barcelona; Spain

3. Department of Surgery, Liver Transplant Unit; University Hospital of Bellvitge; Barcelona Spain

4. Pathological Anatomy Service; University Hospital of Bellvitge; Barcelona Spain

5. Department of Physiological Sciences-II; University of Barcelona; Spain

Funder

Ministry of Economy and Competitiveness (MECC), Spain

AGAUR-Generalitat de Catalunya

E.C-M. was the recipient of a predoctoral fellowship from the FPU program, Ministry of Education, Culture and Sport, Spain

Publisher

Wiley

Subject

Hepatology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/hep.26597/fullpdf

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4. Involvement of EGF receptor and c-Src in the survival signals induced by TGF-beta1 in hepatocytes;Murillo;Oncogene,2005

5. Hepatocytes convert to a fibroblastoid phenotype through the cooperation of TGF-beta1 and Ha-Ras: steps towards invasiveness;Gotzmann;J Cell Sci,2002

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