Pathogenic potentials of glycoprotein C-negative syncytial mutants from rabbit T cells infected persistently with herpes simplex virus type 1-Reference-Cited by-同舟云学术

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Pathogenic potentials of glycoprotein C-negative syncytial mutants from rabbit T cells infected persistently with herpes simplex virus type 1

Published:2005 Issue:1 Volume:76 Page:89-97
ISSN:0146-6615
Container-title:Journal of Medical Virology
language:en
Short-container-title:J. Med. Virol.

Author:

Ishida Yoshimasa,Okabe Tomoyuki,Azukizawa Yoshimi,Isono Takahiro,Seto Akira

Publisher

Wiley

Subject

Infectious Diseases,Virology

Reference36 articles.

1. Truncation of the carboxy-terminal 28 amino acids of glycoprotein B specified by herpes simplex virus type 1 mutant amb1511-7 causes extensive cell fusion

2. Detection of herpes simplex virus DNA sequences in human blood and bone marrow cells

3. Two Novel Single Amino Acid Syncytial Mutations in the Carboxy Terminus of Glycoprotein B of Herpes Simplex Virus Type 1 Confer a Unique Pathogenic Phenotype

4. 1975. Handbook of genetically standardized JAX rabbits. Bar Harbor, Maine: Jackson Laboratory.

5. Glycoprotein C of herpes simplex virus 1 acts as a receptor for the C3b complement component on infected cells

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