Hypophosphatemia as an Early Metabolic Bone Disease Marker in Extremely Low‐Birth‐Weight Infants After Prolonged Parenteral Nutrition Exposure
Author:
Affiliation:
1. Department of Pediatrics Fu Jen Catholic University Hospital Taipei Taiwan
2. Department of Pediatrics National Taiwan University Hospital National Taiwan University College of Medicine Taipei Taiwan
Publisher
Wiley
Subject
Nutrition and Dietetics,Medicine (miscellaneous)
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/jpen.2010
Reference36 articles.
1. Global, regional, and national causes of under-5 mortality in 2000–15: an updated systematic analysis with implications for the Sustainable Development Goals
2. Metabolic bone disease in the preterm infant: Current state and future directions
3. High frequencies of elevated alkaline phosphatase activity and rickets exist in extremely low birth weight infants despite current nutritional support;Mitchell SM;BMC Pediatr,2009
4. Metabolic bone disease of prematurity
5. Parathyroid hormone as a marker for metabolic bone disease of prematurity
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1. Negative Outcomes Associated with Medication in Neonates on Parenteral Nutrition Therapy;Pediatric Reports;2023-06-06
2. Quality improvement project in a neonatal intensive care unit reduced the prevalence and duration of hypophosphatemia with significant and sustainable results;Nutrition in Clinical Practice;2023-04-12
3. Early elevated alkaline phosphatase as a surrogate biomarker of ongoing metabolic bone disease of prematurity;European Journal of Pediatrics;2023-02-16
4. Sodium Glycerophosphate Use in Parenteral Nutrition Improves Mineral Metabolism in Extremely Low Birth Weight Infants;The Journal of Pediatrics;2023-02
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