Mitochondrial toxicity in hearts of CD-1 mice following perinatal exposure to AZT, 3TC, or AZT/3TC in combination-Reference-Cited by-同舟云学术

Mitochondrial toxicity in hearts of CD-1 mice following perinatal exposure to AZT, 3TC, or AZT/3TC in combination

Published:2007 Issue:3-4 Volume:48 Page:190-200
ISSN:0893-6692
Container-title:Environmental and Molecular Mutagenesis
language:en
Short-container-title:Environ. Mol. Mutagen.

Author:

Chan Sherine S.L.,Santos Janine H.,Meyer Joel N.,Mandavilli Bhaskar S.,Cook Dennis L.,McCash Consuelo L.,Kissling Grace E.,Nyska Abraham,Foley Julie F.,van Houten Bennett,Copeland William C.,Walker Vernon E.,Witt Kristine L.,Bishop Jack B.

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Genetics(clinical),Epidemiology

Reference51 articles.

1. Analysis of Gene-Specific DNA Damage and Repair Using Quantitative Polymerase Chain Reaction

2. Persistent mitochondrial dysfunction in HIV-1-exposed but uninfected infants

3. Mitochondrial Damage Revealed by Morphometric and Semiquantitative Analysis of Mouse Pup Cardiomyocytes Following in Utero and Postnatal Exposure to Zidovudine and Lamivudine

4. Genetic damage detected in CD-1 mouse pups exposed perinatally to 3?-azido-3?-deoxythymidine and dideoxyinosine via maternal dosing, nursing, and direct gavage

5. Persistent mitochondrial dysfunction and perinatal exposure to antiretroviral nucleoside analogues

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