CCL21/CCR7 interaction promotes EMT and enhances the stemness of OSCC via a JAK2/STAT3 signaling pathway-Reference-Cited by-同舟云学术

CCL21/CCR7 interaction promotes EMT and enhances the stemness of OSCC via a JAK2/STAT3 signaling pathway

Published:2020-02-04 Issue:9 Volume:235 Page:5995-6009
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:J Cell Physiol

Author:

Chen Yang¹,Shao Zhe¹²,Jiang Erhui¹,Zhou Xiaocheng¹²,Wang Lin¹,Wang Hui¹,Luo Xinyue¹,Chen Qingli¹,Liu Ke¹²,Shang Zhengjun¹²^ORCID

Affiliation:

1. The State Key Laboratory Breeding Base of Basic Science of Stomatology, School and Hospital of StomatologyHubei Province & Key Laboratory of Oral Biomedicine (Wuhan University)Wuhan China

2. Department of Oral and Maxillofacial Head and Neck Oncology, School and Hospital of StomatologyWuhan UniversityWuhan China

Funder

National Natural Science Foundation of China

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.29525

Reference47 articles.

1. Interplay between CCR7 and Notch1 axes promotes stemness in MMTV-PyMT mammary cancer cells

2. TNF-α derived from M2 tumor-associated macrophages promotes epithelial-mesenchymal transition and cancer stemness through the Wnt/β-catenin pathway in SMMC-7721 hepatocellular carcinoma cells

3. Reassessing Epithelial to Mesenchymal Transition as a Prerequisite for Carcinoma Invasion and Metastasis

4. Expression of the C-C Chemokine Receptor 7 Mediates Metastasis of Breast Cancer to the Lymph Nodes in Mice

5. New insights into the mechanisms of epithelial–mesenchymal transition and implications for cancer

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