Taiwanin C elicits apoptosis in arecoline and 4‐nitroquinoline‐1‐oxide‐induced oral squamous cell carcinoma cells and hinders proliferation via epidermal growth factor receptor/PI3K suppression-Reference-Cited by-同舟云学术

Taiwanin C elicits apoptosis in arecoline and 4‐nitroquinoline‐1‐oxide‐induced oral squamous cell carcinoma cells and hinders proliferation via epidermal growth factor receptor/PI3K suppression

Published:2019-03-18 Issue:6 Volume:34 Page:760-767
ISSN:1520-4081
Container-title:Environmental Toxicology
language:en
Short-container-title:Environmental Toxicology

Author:

Tsai Cheng‐Yen¹,Fang Hsin‐Yuan²,Shibu Marthandam Asokan³,Lin Yueh‐Min⁴,Chou Yung‐Chen⁵,Chen Yi‐Hui⁶,Day Cecilia‐Hsuan⁷,Shen Chia‐Yao⁷,Ban Bo⁸,Huang Chih‐Yang³⁹¹⁰¹¹^ORCID

Affiliation:

1. Department of PediatricsChina Medical University Beigang Hospital Yunlin Taiwan

2. Department of Thoracic SurgeryChina Medical University Hospital Taichung Taiwan

3. Medical Research Center for Exosomes and Mitochondria Related DiseasesChina Medical University Hospital Taichung Taiwan

4. Department of PathologyChanghua Christian Hospital Changhua Taiwan

5. Department of Computer Science and Information EngineeringAsia University Taichung Taiwan

6. Department of M‐Commerce and Multimedia ApplicationsAsia University Taichung Taiwan

7. Department of NursingMeiho University Pingtung Taiwan

8. Department of EndocrinologyAffiliated Hospital of Jining Medical University, Jining Medical University Jining Shandong China

9. Graduate Institute of Chinese Medical ScienceChina Medical University Taichung Taiwan

10. Department of BiotechnologyAsia University Taichung Taiwan

11. Graduate Institute of Basic Medical ScienceChina Medical University Taichung Taiwan

Publisher

Wiley

Subject

Health, Toxicology and Mutagenesis,Management, Monitoring, Policy and Law,Toxicology,General Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/tox.22742

Reference42 articles.

1. Gene expression in human oral squamous cell carcinoma is influenced by risk factor exposure

2. Transactivation and expression patterns of Jun and Fos/AP-1 super-family proteins in human oral cancer

3. Ras oncogenes in oral cancer: The past 20 years

4. Lifestyle risk factors for oral cancer

5. Betel quid chewing, cigarette smoking and alcohol consumption related to oral cancer in Taiwan

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