Discovery and Function of the HGF/MET and the MSP/RON Kinase Signaling Pathways in Cancer

Author:

Benvenuti Silvia1,Milan Melissa1,Comoglio Paolo M.1

Affiliation:

1. Candiolo Cancer Institute; Italy

Publisher

John Wiley & Sons, Ltd

Reference209 articles.

1. The MET receptor tyrosine kinase in invasion and metastasis;Benvenuti;J Cell Physiol,2007

2. Scatter-factor and semaphorin receptors: cell signalling for invasive growth;Trusolino;Nature Reviews,2002

3. Met/Hepatocyte growth factor receptor ubiquitination suppresses transformation and is required for Hrs phosphorylation;Abella;Molecular and Cellular Biology,2005

4. Mutation of the c-Cbl TKB domain binding site on the Met receptor tyrosine kinase converts it into a transforming protein;Peschard;Mol Cell,2001

5. Specific uncoupling of GRB2 from the Met receptor. Differential effects on transformation and motility;Ponzetto;The Journal of Biological Chemistry,1996

Cited by 1 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献

1. Role of HGF/c‐Met in the treatment of colorectal cancer with liver metastasis;Journal of Biochemical and Molecular Toxicology;2019-03-21

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