Bik subcellular localization in response to oxidative stress induced by chemotherapy, in Two different breast cancer cell lines and a Non-tumorigenic epithelial cell line

Author:

Trejo-Vargas Aglaé123,Hernández-Mercado Elisa12,Ordóñez-Razo Rosa María2,Lazzarini Roberto1,Arenas-Aranda Diego Julio2,Gutiérrez-Ruiz María Concepción1,Königsberg Mina1,Luna-López Armando4

Affiliation:

1. Departamento de Ciencias de la Salud, DCBS; Universidad Autónoma Metropolitana Iztapalapa; México D.F. México

2. Unidad Médica en Genética Humana; Centro Médico Nacional Siglo XXI; México D.F. Mexico

3. Posgrado en Biologia Experimental

4. Instituto Nacional de Geriatria, SSA; Posgrado en Biologia Experimental, UAMI; México D.F. Mexico

Funder

CBS-UAMI

CONACyT

CONACyT and INGER DI

Publisher

Wiley

Subject

Toxicology

Reference57 articles.

1. Primary cultured astrocytes from old rats are capable to activate the Nrf2 response against MPP+ toxicity after tBHQ pre-treatment;Alarcón-Aguilar;Neurobiol. Aging,2014

2. Intraperitoneal Cisplatin and Paclitaxel in ovarian cancer;Armstrong;N. Engl. J. Med.,2006

3. Neo-adjuvant capecitabine chemotherapy in women with newly diagnosed locally advanced breast cancer in a resource-poor setting (Nigeria): efficacy and safety in a phase II feasibility study;Arowolo;Breast J.,2013

4. Targeting extracellular ROS signaling of tumor cells;Bauer;Anticancer Res,2014

5. BH3-only protein Bik is involved in both apoptosis induction and sensitivity to oxidative stress in multiple myeloma;Bodet;Br. J. Cancer,2010

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