New interactions between tumor suppressor Fhit protein and a nonhydrolyzable analog of its AP4A substrate-Reference-Cited by-同舟云学术

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New interactions between tumor suppressor Fhit protein and a nonhydrolyzable analog of its AP4A substrate

Published:2017-02 Issue:3 Volume:591 Page:548-559
ISSN:0014-5793
Container-title:FEBS Letters
language:en
Short-container-title:FEBS Lett

Author:

Krakowiak Agnieszka¹,Kocoń-Rębowska Beata¹,Dolot Rafał¹,Piotrzkowska Danuta¹

Affiliation:

1. Department of Bioorganic Chemistry; Polish Academy of Sciences, Centre of Molecular and Macromolecular Studies; Lodz Poland

Funder

Polish Ministry of Science and Higher Education

The National Science Centre

Publisher

Wiley

Subject

Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics

Reference49 articles.

1. The tumor-suppressor gene FHIT is involved in the regulation of apoptosis and in cell cycle control;Sard;Proc Natl Acad Sci USA,1999

2. A. Fhit-ing role in the DNA damage checkpoint response;Ishii;Cell Cycle,2007

3. Fragile gene product, Fhit, in oxidative and replicative stress responses;Okumura;Cancer Sci,2009

4. Pathology and biology associated with the fragile FHIT Gene and gene product;Saldivar;J Cell Biochem,2010

5. Fhit interaction with ferredoxin reductase triggers generation of reactive oxygen species and apoptosis of cancer cells;Trapasso;J Biol Chem,2008

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Biochemical, crystallographic and biophysical characterization of histidine triad nucleotide-binding protein 2 with different ligands including a non-hydrolyzable analog of Ap4A;Biochimica et Biophysica Acta (BBA) - General Subjects;2021-11

2. Tanshinones induce tumor cell apoptosis via directly targeting FHIT;Scientific Reports;2021-06-09

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