Middle East respiratory syndrome coronavirus ORF4b protein inhibits TLR7 ‐ and TLR9 ‐dependent alpha interferon induction
Author:
Affiliation:
1. Division of Microbiology and Infectious Diseases, Department of Pathology Shiga University of Medical Science Otsu Japan
2. Department of Microbiology, Faculty of Bio‐Science Nagahama Institute of Bio‐Science and Technology Japan
Funder
Japan Society for the Promotion of Science
Shiga University of Medical Science
Publisher
Wiley
Subject
Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/1873-3468.14486
Reference42 articles.
1. The structural and accessory proteins M, ORF 4a, ORF 4b, and ORF 5 of Middle East respiratory syndrome coronavirus (MERS-CoV) are potent interferon antagonists
2. The ORF4b-encoded accessory proteins of Middle East respiratory syndrome coronavirus and two related bat coronaviruses localize to the nucleus and inhibit innate immune signalling
3. Control of Innate Immune Activation by Severe Acute Respiratory Syndrome Coronavirus 2 and Other Coronaviruses
4. Innate Immunity Evasion Strategies of Highly Pathogenic Coronaviruses: SARS-CoV, MERS-CoV, and SARS-CoV-2
5. MERS-CoV Accessory ORFs Play Key Role for Infection and Pathogenesis
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1. Recurrent viral capture of cellular phosphodiesterases that antagonize OAS-RNase L;Proceedings of the National Academy of Sciences;2024-01-26
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