Secretion of IL-1β from imatinib-resistant chronic myeloid leukemia cells contributes toBCR-ABLmutation-independent imatinib resistance-Reference-Cited by-同舟云学术

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Secretion of IL-1β from imatinib-resistant chronic myeloid leukemia cells contributes toBCR-ABLmutation-independent imatinib resistance

Published:2016-01-25 Issue:3 Volume:590 Page:358-368
ISSN:0014-5793
Container-title:FEBS Letters
language:en
Short-container-title:FEBS Lett

Author:

Lee Cho-Rong¹,Kang Jung-Ah¹,Kim Hye-Eun¹,Choi Yegyun¹,Yang Taewoo¹,Park Sung-Gyoo¹

Affiliation:

1. School of Life Sciences; Gwangju Institute of Science and Technology; Korea

Funder

Korean Health Technology R&D Project

Korea Health Industry Development Institute

Ministry of Health and Welfare, Republic of Korea

Gwangju Institute of Science and Technology

Publisher

Wiley

Subject

Cell Biology,Genetics,Molecular Biology,Biochemistry,Structural Biology,Biophysics

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/1873-3468.12057/fullpdf

Reference49 articles.

1. Frequency of major molecular responses to imatinib or interferon alfa plus cytarabine in newly diagnosed chronic myeloid leukemia;Hughes;N Engl J Med,2003

2. Philadelphia chromosome-positive leukemias: from basic mechanisms to molecular therapeutics;Kurzrock;Ann Intern Med,2003

3. The molecular biology of chronic myeloid leukaemia;Melo;Leukemia,1996

4. Regulation of the c-Abl and Bcr-Abl tyrosine kinases;Hantschel;Nat Rev Mol Cell Biol,2004

5. TLS/FUS, a pro-oncogene involved in multiple chromosomal translocations, is a novel regulator of BCR/ABL-mediated leukemogenesis;Perrotti;EMBO J,1998

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