Brief Report: Connecting Two Pathways Through Ca2+Signaling: NLRP3 Inflammasome Activation Induced by a HypermorphicPLCG2Mutation
Author:
Affiliation:
1. National Human Genome Research Institute, NIHBethesdaMaryland
2. National Institute of Allergy and Infectious Diseases, NIHBethesdaMaryland
Publisher
Wiley
Subject
Immunology,Rheumatology,Immunology and Allergy
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/art.38961
Reference12 articles.
1. Review: Beyond the NLRP3 Inflammasome: Autoinflammatory Diseases Reach Adolescence
2. A Hypermorphic Missense Mutation in PLCG2 , Encoding Phospholipase Cγ2, Causes a Dominantly Inherited Autoinflammatory Disease with Immunodeficiency
3. Biologic basis for interleukin‐1 in disease;Dinarello CA;Blood,1996
4. The Inflammasomes
5. Mutation of a new gene encoding a putative pyrin-like protein causes familial cold autoinflammatory syndrome and Muckle–Wells syndrome
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