Insulin deficiency, but not resistance, exaggerates cognitive deficits in transgenic mice expressing human amyloid and tau proteins. Reversal by Exendin‐4 treatment-Reference-Cited by-同舟云学术

Insulin deficiency, but not resistance, exaggerates cognitive deficits in transgenic mice expressing human amyloid and tau proteins. Reversal by Exendin‐4 treatment

Published:2020-08 Issue:11 Volume:98 Page:2357-2369
ISSN:0360-4012
Container-title:Journal of Neuroscience Research
language:en
Short-container-title:J Neurosci Res

Author:

King Matthew R.¹,Anderson Nicholas J.¹,Deciu Mihaela¹,Guernsey Lucie S.¹,Cundiff Morgan¹,Hajizadeh Shohreh¹,Jolivalt Corinne G.¹^ORCID

Affiliation:

1. Department of Pathology University of California San Diego La Jolla CA USA

Funder

National Institutes of Health

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jnr.24706

Reference78 articles.

1. Synaptic plasticity in the diabetic brain: advanced aging?

2. Diabetes Mellitus and Risk of Alzheimer Disease and Decline in Cognitive Function

3. Iso-α-acids and matured hop bitter acids in beer improve obesity-induced cognitive impairment

4. The diabetes drug liraglutide reverses cognitive impairment in mice and attenuates insulin receptor and synaptic pathology in a non-human primate model of Alzheimer's disease

5. Glucagon-like peptide 1 improved glycemic control in type 1 diabetes

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