Abnormal degradative pathway of mitochondrial ATP synthase subunit c in late infantile neuronal ceroid-lipofuscinosis (Batten disease)-Reference-Cited by-同舟云学术

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Abnormal degradative pathway of mitochondrial ATP synthase subunit c in late infantile neuronal ceroid-lipofuscinosis (Batten disease)

Published:1995-06 Issue:2 Volume:57 Page:254-259
ISSN:0148-7299
Container-title:American Journal of Medical Genetics
language:en
Short-container-title:Am. J. Med. Genet.

Author:

Ezaki Junji,Wolfe Leonhard S.,Ishidoh Kazumi,Kominami Eiki

Publisher

Wiley

Subject

Genetics(clinical)

Reference26 articles.

1. Liver mitochondria contain an ATP-dependent, vanadate-sensitive pathway for the degradation of proteins.

2. Sequences of members of the human gene family for the c subunit of mitochondrial ATP synthase

3. DNA sequences of a bovine gene and of two related pseudogenes for the proteolipid subunit of mitochondrial ATP synthase

4. Molecular basis of lysosomal accumulation of subunit c of mitochondrial ATP synthase in neuronal ceroid-lipofuscinosis

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2. Cellular Pathology of Lysosomal Storage Disorders;Brain Pathology;2006-04-05

3. The Neuronal Ceroid-Lipofuscinoses. Recent Advances;Brain Pathology;2006-04-05

4. Defective lysosomal arginine transport in juvenile Batten disease;Human Molecular Genetics;2005-10-26

5. Sex-specific chromosome instability in early human development;American Journal of Medical Genetics Part A;2005

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