Yersinia pestisAil recruitment of C4b-binding protein leads to factor I-mediated inactivation of covalently and noncovalently bound C4b-Reference-Cited by-同舟云学术

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Yersinia pestisAil recruitment of C4b-binding protein leads to factor I-mediated inactivation of covalently and noncovalently bound C4b

Published:2014-01-13 Issue:3 Volume:44 Page:742-751
ISSN:0014-2980
Container-title:European Journal of Immunology
language:en
Short-container-title:Eur. J. Immunol.

Author:

Ho Derek K.¹,Skurnik Mikael¹²,Blom Anna M.³,Meri Seppo¹²

Affiliation:

1. Immunobiology Program, Department of Bacteriology and Immunology; Haartman Institute, University of Helsinki; Helsinki Finland

2. HUSLAB; Helsinki University Central Hospital; Helsinki Finland

3. Division of Medical Protein Chemistry; Lund University Department of Laboratory Medicine; Malmö Sweden

Publisher

Wiley

Subject

Immunology,Immunology and Allergy

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/eji.201343552/fullpdf

Reference32 articles.

1. Plague gives surprises in the first decade of the 21st century in the United States and worldwide;Butler;Am. J. Trop. Med. Hyg.,2013

2. Multidrug resistance in Yersinia pestis mediated by a transferable plasmid;Galimand;N. Engl. J. Med.,1997

3. Plague as a biological weapon: medical and public health management. Working Group on Civilian Biodefense;Inglesby;JAMA,2000

4. Virulence factors of Yersinia pestis are overcome by a strong lipopolysaccharide response;Montminy;Nat. Immunol.,2006

5. Yersinia outer proteins: Yops;Trosky;Cell Microbiol.,2008

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2. Microbial evasion of the complement system: a continuous and evolving story;Frontiers in Immunology;2024-01-04

3. Diverse Functions of C4b-Binding Protein in Health and Disease;The Journal of Immunology;2023-11-15

4. Yersinia pestis Δ ail Mutants Are Not Susceptible to Human Complement Bactericidal Activity in the Flea;Applied and Environmental Microbiology;2023-02-28

5. Lipopolysaccharides and outer membrane proteins as main structures involved in complement evasion strategies of non-typhoidal Salmonella strains;Molecular Immunology;2022-10

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