BACH1 promotes the progression of esophageal squamous cell carcinoma by inducing the epithelial–mesenchymal transition and angiogenesis-Reference-Cited by-同舟云学术

BACH1 promotes the progression of esophageal squamous cell carcinoma by inducing the epithelial–mesenchymal transition and angiogenesis

Published:2021-05 Issue:10 Volume:10 Page:3413-3426
ISSN:2045-7634
Container-title:Cancer Medicine
language:en
Short-container-title:Cancer Med

Author:

Zhao Yan¹^ORCID,Gao Jiajia¹,Xie Xiufeng¹,Nan Peng¹,Liu Fang¹,Sun Yulin¹,Zhao Xiaohang¹^ORCID

Affiliation:

1. State Key Laboratory of Molecular Oncology, National Cancer Center/National Clinical Research Center for Cancer/Cancer Hospital Chinese Academy of Medical Sciences and Peking Union Medical College Beijing China

Publisher

Wiley

Subject

Cancer Research,Radiology Nuclear Medicine and imaging,Oncology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/cam4.3884

Reference48 articles.

1. Global cancer statistics 2018: GLOBOCAN estimates of incidence and mortality worldwide for 36 cancers in 185 countries

2. Oesophageal cancer

3. Bach1: Function, Regulation, and Involvement in Disease

4. BACH1, the master regulator gene: A novel candidate target for cancer therapy

5. The Bach Family of Transcription Factors: A Comprehensive Review

Cited by 19 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Thermosensitive gel-nano system against esophageal cancer via restoring p53 activity and boosting T-cell immunity;Journal of Controlled Release;2024-07

2. Type I Diabetes Mellitus Suppresses Experimental Skin Carcinogenesis;Cancers;2024-04-15

3. Transcription factor BACH1 in cancer: roles, mechanisms, and prospects for targeted therapy;Biomarker Research;2024-02-07

4. Modern representations about signaling pathways and protective mechanisms of ferroptosis. A biological role of diffusion of death signals of ferroptotic cells;Reviews on Clinical Pharmacology and Drug Therapy;2023-10-10

5. BACH1 Loss Exerts Antitumor Effects on Mantle Cell Lymphoma Cells via Inducing a Tumor-Intrinsic Innate Immune Response and Cell-Cycle Arrest;Molecular Cancer Research;2023-09-15