Loss of stimulatory effect of guanosine triphosphate on [35S]GTPγS binding correlates with Alzheimer's disease neurofibrillary pathology in entorhinal cortex and CA1 hippocampal subfield-Reference-Cited by-同舟云学术

Loss of stimulatory effect of guanosine triphosphate on [35S]GTPγS binding correlates with Alzheimer's disease neurofibrillary pathology in entorhinal cortex and CA1 hippocampal subfield

Published:2001-12-21 Issue:3 Volume:67 Page:388-398
ISSN:0360-4012
Container-title:Journal of Neuroscience Research
language:en
Short-container-title:J. Neurosci. Res.

Author:

García-Jiménez A.,Cowburn R.F.,Ohm T.G.,Lasn H.,Winblad B.,Bogdanovic N.,Fastbom J.

Funder

Karolinska Institutet Research Fund

Sandoz Foundation for Gerontological Research

Gamla Tjänarinnor Foundation

Swedish Medical Research Council

Swedish Alzheimer's Disease Fund

The Swedish Society of Medicine

Sigurd and Elsa Golje's Foundation

Loo and Hans Osterman's Foundation

Gun and Bertil Stohne's Foundation

Augusta and Petrus Hedlund's Foundation

Magnus Bergvalls and Åke Wiberg's Foundation

Riksbankens Jubileumsfond

Deutsche Forschungsgemeinschaft

Hertie-Stiftung

European Union Biomed-2 Concerted Action

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience

Reference39 articles.

1. Hyperphosphorylation of human tau by brain kinase PK40erk beyond phosphorylation by cAMP-dependent PKA: relation to Alzheimer's disease;Blanchard;Biochem Biophys Res Commun,1994

2. A quantitative autoradiographic study of [3H]cAMP binding to cytosolic and particulate protein kinase A in post-mortem brain staged for Alzheimer's disease neurofibrillary changes and amyloid deposits;Bonkale;Brain Res,1999

3. Neuropathological stageing of Alzheimer-related changes;Braak;Acta Neuropathol,1991

4. Calcium regulates processing of the Alzheimer amyloid protein precursor in a protein kinase C-independent manner;Buxbaum;Proc Natl Acad Sci USA,1994

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