Flightless I deficiency enhances wound repair by increasing cell migration and proliferation-Reference-Cited by-同舟云学术

Flightless I deficiency enhances wound repair by increasing cell migration and proliferation

Published:2007-02-26 Issue:5 Volume:211 Page:572-581
ISSN:0022-3417
Container-title:The Journal of Pathology
language:en
Short-container-title:J. Pathol.

Author:

Cowin AJ¹²³,Adams DH¹,Strudwick XL¹,Chan H¹,Hooper JA⁴,Sander GR¹²,Rayner TE¹,Matthaei KI⁵,Powell BC¹²³,Campbell HD⁴

Affiliation:

1. Child Health Research Institute, 72 King William Road, North Adelaide, South Australia

2. Department of Paediatrics, University of Adelaide, South Australia

3. School of Pharmacy and Medical Sciences, University of South Australia, Australia

4. Molecular Genetics and Evolution Group and Centre for the Molecular Genetics of Development, Research School of Biological Sciences, Australian National University, Canberra, ACT, Australia

5. Division of Molecular Bioscience, The John Curtin School of Medical Research, Canberra, ACT, Australia

Publisher

Wiley

Subject

Pathology and Forensic Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/path.2143

Reference23 articles.

1. Gelsolin, a Multifunctional Actin Regulatory Protein

2. Mechanisms of epithelial fusion and repair

3. Differential Effect of Wounding on Actin and its Associated Proteins, Paxillin and Gelsolin, in Fetal Skin Explants

4. Functions of gelsolin: motility, signaling, apoptosis, cancer

5. Characterization of a Novel Ras-Binding Protein Ce-FLI-1 Comprising Leucine-Rich Repeats and Gelsolin-like Domains

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