PDK4‐Deficiency Reprograms Intrahepatic Glucose and Lipid Metabolism to Facilitate Liver Regeneration in Mice
Author:
Affiliation:
1. Department of Physiology & Neurobiology University of Connecticut Storrs CT
2. Department of Internal Medicine Section of Digestive Diseases Yale University New Haven CT
Funder
National Institute on Alcohol Abuse and Alcoholism
Publisher
Wiley
Subject
Hepatology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/hep4.1484
Reference46 articles.
1. Liver regeneration
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4. Farnesoid X receptor activation promotes cell proliferation via PDK4-controlled metabolic reprogramming
5. Loss of the novel mitochondrial protein FAM210B promotes metastasis via PDK4-dependent metabolic reprogramming
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