Vitamin D3 ameliorates nitrogen mustard‐induced cutaneous inflammation by inactivating the NLRP3 inflammasome through the SIRT3–SOD2–mtROS signaling pathway-Reference-Cited by-同舟云学术

Vitamin D3 ameliorates nitrogen mustard‐induced cutaneous inflammation by inactivating the NLRP3 inflammasome through the SIRT3–SOD2–mtROS signaling pathway

Published:2021-02 Issue:2 Volume:11 Page:
ISSN:2001-1326
Container-title:Clinical and Translational Medicine
language:en
Short-container-title:Clinical and Translational Medicine

Author:

Dong Xunhu¹²,He Ying³,Ye Feng¹²,Zhao Yuanpeng¹²,Cheng Jin¹²,Xiao Jingsong¹²,Yu Wenpei¹²,Zhao Jiqing¹²,Sai Yan¹²,Dan Guorong¹²,Chen Mingliang¹²⁴,Zou Zhongmin¹²

Affiliation:

1. Department of Chemical Defense Medicine, School of Military Preventive Medicine Third Military Medical University (Army Medical University) Chongqing China

2. Institute of Toxicology, School of Military Preventive Medicine Third Military Medical University (Army Medical University) Chongqing China

3. Department of Ultrasound Xinqiao Hospital Third Military Medical University (Army Medical University) Chongqing China

4. Institute of Pathology and Southwest Cancer Centre, Southwest Hospital Third Military Medical University (Army Medical University) Chongqing China

Funder

National Natural Science Foundation of China

National Postdoctoral Program for Innovative Talents

Publisher

Wiley

Subject

Molecular Medicine,Medicine (miscellaneous)

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ctm2.312

Reference53 articles.

1. Molecular toxicology of sulfur mustard-induced cutaneous inflammation and blistering

2. Nitrogen mustard exposure of murine skin induces DNA damage, oxidative stress and activation of MAPK/Akt-AP1 pathway leading to induction of inflammatory and proteolytic mediators

3. Absence of a p53 allele delays nitrogen mustard-induced early apoptosis and inflammation of murine skin

4. Mitigation of nitrogen mustard mediated skin injury by a novel indomethacin bifunctional prodrug

5. Limitations and challenges in treatment of acute chemical warfare agent poisoning

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