Receptor interacting protein kinase 1 mediates murine acetaminophen toxicity independent of the necrosome and not through necroptosis-Reference-Cited by-同舟云学术

Receptor interacting protein kinase 1 mediates murine acetaminophen toxicity independent of the necrosome and not through necroptosis

Published:2015-07-31 Issue:6 Volume:62 Page:1847-1857
ISSN:0270-9139
Container-title:Hepatology
language:en
Short-container-title:Hepatology

Author:

Dara Lily¹,Johnson Heather¹,Suda Jo¹,Win Sanda¹,Gaarde William²,Han Derick¹³,Kaplowitz Neil¹

Affiliation:

1. University of Southern California Research Center for Liver Diseases, and the Division of GI-Liver, Department of Medicine, Keck School of Medicine, University of Southern California; Los Angeles CA

2. ISIS Pharmaceuticals; Carlsbad CA

3. Department of Biopharmaceutical Sciences; School of Pharmacy, Keck Graduate Institute; Claremont CA

Funder

National Institute of Diabetes and Digestive and Kidney Diseases

Publisher

Wiley

Subject

Hepatology

Reference52 articles.

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2. Acetaminophen hepatotoxicity in tumor necrosis factor/lymphotoxin-alpha gene knockout mice;Boess;Hepatology,1998

3. Role of caspases in acetaminophen-induced liver injury;Jaeschke;Life Sci,2006

4. The mechanism underlying acetaminophen-induced hepatotoxicity in humans and mice involves mitochondrial damage and nuclear DNA fragmentation;McGill;J Clin Invest,2012

5. No evidence for caspase-dependent apoptosis in acetaminophen hepatotoxicity;Jaeschke;Hepatology,2011

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