LL‐37 and its analog FF/CAP18 attenuate neutrophil migration in sepsis‐induced acute lung injury-Reference-Cited by-同舟云学术

LL‐37 and its analog FF/CAP18 attenuate neutrophil migration in sepsis‐induced acute lung injury

Published:2018-12-09 Issue:4 Volume:120 Page:4863-4871
ISSN:0730-2312
Container-title:Journal of Cellular Biochemistry
language:en
Short-container-title:J Cell Biochem

Author:

Qin Xiuchuan¹²,Zhu Guangfa¹^ORCID,Huang Lixue¹,Zhang Wenwei¹,Huang Yan¹,Xi Xin¹

Affiliation:

1. Department of Pulmonary and Critical Care Medicine Beijing Anzhen Hospital, Capital Medical University, Experimenter Center, Beijing Institute of Heart, Lung and Blood Vessel Diseases Beijing China

2. Emergency and Critical Care Center, Beijing Anzhen Hospital, Capital Medical University, Beijing Institute of Heart, Lung and Blood Vessel Diseases Beijing China

Publisher

Wiley

Subject

Cell Biology,Molecular Biology,Biochemistry

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcb.27641

Reference41 articles.

1. Consensus conference definitions for sepsis, septic shock, acute lung injury, and acute respiratory distress syndrome: Time for a reevaluation

2. Endogenous antimicrobial peptide LL-37 induces human vasodilatation † †A preliminary account of these results has been presented to the 29th Congress of the Scandinavian Society of Anaesthesiology and Intensive Care, Gothenburg, September 5–8, 2007.

3. The Human Cationic Peptide LL-37 Induces Activation of the Extracellular Signal-Regulated Kinase and p38 Kinase Pathways in Primary Human Monocytes

4. Cathelicidin LL-37: A Multitask Antimicrobial Peptide

5. Severe Sepsis and Septic Shock: Clinical Overview and Update on Management

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