Dysregulated ΔNp63α negatively regulates the maspin promoter in keratinocytes via blocking endogenous p73 binding
Author:
Affiliation:
1. Office of Biotechnology Products; CDER/FDA; Bethesda Maryland
2. National Cancer Institute; NIH; Bethesda Maryland
Publisher
Wiley
Subject
Cancer Research,Molecular Biology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/mc.22022/fullpdf
Reference121 articles.
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3. An anti-apoptotic role for the p53 family member, p73, during developmental neuron death;Pozniak;Science,2000
4. Identification and functional characterization of two new transcriptional variants of the human p63 gene;Mangiulli;Nucleic Acids Res,2009
5. Additional complexity in p73: Induction by mitogens in lymphoid cells and identification of two new splicing variants epsilon and zeta;De Laurenzi;Cell Death Differ,1999
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1. CDK1 Promotes Epithelial–Mesenchymal Transition and Migration of Head and Neck Squamous Carcinoma Cells by Repressing ∆Np63α-Mediated Transcriptional Regulation;International Journal of Molecular Sciences;2022-07-02
2. Maspin, Syndecan-1, and Ki-67 in the Odontogenic Keratocyst: An Immunohistochemical Analysis;International Journal of Dentistry;2020-07-14
3. p53-Related Transcription Targets of TAp73 in Cancer Cells—Bona Fide or Distorted Reality?;International Journal of Molecular Sciences;2020-02-17
4. Tissue inhibitor of matrix metalloproteinase-3 has both anti-metastatic and anti-tumourigenic properties;Clinical & Experimental Metastasis;2020-01-01
5. Molecular Mechanisms of p63-Mediated Squamous Cancer Pathogenesis;International Journal of Molecular Sciences;2019-07-23
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