Silencing of lipocalin‐2 improves cardiomyocyte viability under iron overload conditions via decreasing mitochondrial dysfunction and apoptosis-Reference-Cited by-同舟云学术

Silencing of lipocalin‐2 improves cardiomyocyte viability under iron overload conditions via decreasing mitochondrial dysfunction and apoptosis

Author:

Kumfu Sirinart¹²³,Siri‐Angkul Natthaphat¹²³,Chattipakorn Siriporn C.¹³,Chattipakorn Nipon¹²³^ORCID

Affiliation:

1. Faculty of Medicine, Cardiac Electrophysiology Research and Training Center Chiang Mai University Chiang Mai Thailand

2. Cardiac Electrophysiology Unit, Department of Physiology Chiang Mai University Chiang Mai Thailand

3. Center of Excellence in Cardiac Electrophysiology Research Chiang Mai University Chiang Mai Thailand

Funder

National Research Council of Thailand

National Science and Technology Development Agency

Thailand Research Fund

Chiang Mai University

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Link

Reference77 articles.

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