BCL11B promotes T‐cell acute lymphoblastic leukaemia cell survival via the XRCC5/C11ORF21 axis-Reference-Cited by-同舟云学术

BCL11B promotes T‐cell acute lymphoblastic leukaemia cell survival via the XRCC5/C11ORF21 axis

Published:2024-02 Issue:2 Volume:14 Page:
ISSN:2001-1326
Container-title:Clinical and Translational Medicine
language:en
Short-container-title:Clinical & Translational Med

Author:

Yu Xibao¹²³^ORCID,Li Yuchen¹²,Yang Pengyue¹²,Wang Yan¹²,Liu Xuan¹²,Cai Letong¹²,Lai Jing¹,Zhang Yue¹,Zha Xianfeng⁴,Przybylski Grzegorz Krzysztof⁵,Xu Ling¹²^ORCID,Li Yangqiu¹²^ORCID

Affiliation:

1. The First Affiliated Hospital and Institute of Hematology, School of Medicine Jinan University Guangzhou Guangdong China

2. Key Laboratory for Regenerative Medicine of Ministry of Education Jinan University Guangzhou Guangdong China

3. Guangzhou Municipality Tianhe Nuoya Bio‐Engineering Co. Ltd., Guangzhou 510663 Guangdong China

4. Department of Clinical Laboratory, First Affiliated Hospital Jinan University Guangzhou Guangdong China

5. Institute of Human Genetics Polish Academy of Sciences Poznan Poland

Funder

National Natural Science Foundation of China

Publisher

Wiley

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ctm2.1580

Reference10 articles.

1. Bcl11b and combinatorial resolution of cell fate in the T-cell gene regulatory network

2. Reprogramming of T Cells to Natural Killer–Like Cells upon Bcl11b Deletion

3. Inhibition of BCL11B induces downregulation of PTK7 and results in growth retardation and apoptosis in T-cell acute lymphoblastic leukemia

4. Integrated genomic analyses identify high-risk factors and actionable targets in T-cell acute lymphoblastic leukemia

5. Ku80 is highly expressed in lung adenocarcinoma and promotes cisplatin resistance