Kif1B? isoform is enriched in motor neurons but does not change in a mouse model of amyotrophic lateral sclerosis-Reference-Cited by-同舟云学术

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Kif1B? isoform is enriched in motor neurons but does not change in a mouse model of amyotrophic lateral sclerosis

Published:2003-02-12 Issue:5 Volume:71 Page:732-739
ISSN:0360-4012
Container-title:Journal of Neuroscience Research
language:en
Short-container-title:J. Neurosci. Res.

Author:

Conforti Laura,Dell'Agnello Carlotta,Calvaresi Novella,Tortarolo Massimo,Giorgini Andrea,Coleman Michael P.,Bendotti Caterina

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience

Reference29 articles.

1. Kinesin family in murine central nervous system.

2. Transgenic SOD1 G93A mice develop reduced GLT-1 in spinal cord without alterations in cerebrospinal fluid glutamate levels

3. Fast axonal transport alterations in amyotrophic lateral sclerosis (ALS) and in parathyroid hormone (PTH)-treated axons

4. Defective axonal transport in a transgenic mouse model of amyotrophic lateral sclerosis

5. The major brain isoform of Kif1b lacks the putative mitochondria-binding domain

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1. Genome-Wide and Transcriptome-Wide Association Studies on Northern New England and Ohio Amyotrophic Lateral Sclerosis Cohorts;Neurology Genetics;2024-10

2. Partitioning RNAs by length improves transcriptome reconstruction from short-read RNA-seq data;Nature Biotechnology;2022-01-10

3. Cytoskeletal regulation guides neuronal trafficking to effectively supply the synapse;Current Biology;2021-05

4. Circulating RNAs as Potential Biomarkers in Amyotrophic Lateral Sclerosis;International Journal of Molecular Sciences;2020-03-03

5. Postsynaptic density protein 95 (PSD-95) is transported by KIF5 to dendritic regions;Molecular Brain;2019-11-21

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