Spinal release of tumour necrosis factor activates c‐ J un N ‐terminal kinase and mediates inflammation‐induced hypersensitivity-Reference-Cited by-同舟云学术

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Spinal release of tumour necrosis factor activates c‐ J un N ‐terminal kinase and mediates inflammation‐induced hypersensitivity

Published:2014-06-18 Issue:2 Volume:19 Page:260-270
ISSN:1090-3801
Container-title:European Journal of Pain
language:en
Short-container-title:EJP

Author:

Bas D.B.¹,Abdelmoaty S.¹,Sandor K.¹,Codeluppi S.²,Fitzsimmons B.³,Steinauer J.³,Hua X.Y.³,Yaksh T.L.³,Svensson C.I.¹

Affiliation:

1. Department of Physiology and Pharmacology Karolinska Institutet Stockholm Sweden

2. Department of Medical Biochemistry and Biophysics Karolinska Institutet Stockholm Sweden

3. Department of Anesthesiology University of California San Diego La Jolla USA

Funder

Swedish Foundation for Strategic Research

National Institutes of Health

Publisher

Wiley

Subject

Anesthesiology and Pain Medicine

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/ejp.544

Reference50 articles.

1. Adjuvant-induced arthritis: IL-1β, IL-6 and TNF-α are up-regulated in the spinal cord

2. Collagen antibody-induced arthritis evokes persistent pain with spinal glial involvement and transient prostaglandin dependency

3. Increased Tumor Necrosis Factor-α and Prostaglandin E2 Concentrations in the Cerebrospinal Fluid of Rats with Inflammatory Hyperalgesia: The Effects of Analgesic Drugs

4. Spinal tumor necrosis factor α neutralization reduces peripheral inflammation and hyperalgesia and suppresses autonomic responses in experimental arthritis: A role for spinal tumor necrosis factor α during induction and maintenance of peripheral inflammat

5. Regulation of Peripheral Inflammation by Spinal p38 MAP Kinase in Rats

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