<i>PNPLA3</i> rs738409 and risk of fibrosis in NAFLD: Exploring mediation pathways through intermediate histological features-Reference-Cited by-同舟云学术

PNPLA3 rs738409 and risk of fibrosis in NAFLD: Exploring mediation pathways through intermediate histological features

Published:2022-04-21 Issue:5 Volume:76 Page:1482-1494
ISSN:0270-9139
Container-title:Hepatology
language:en
Short-container-title:Hepatology

Author:

Vilar‐Gomez Eduardo¹^ORCID,Pirola Carlos J.²^ORCID,Sookoian Silvia²^ORCID,Wilson Laura A.³,Liang Tiebing¹,Chalasani Naga¹^ORCID

Affiliation:

1. Division of Gastroenterology and Hepatology Department of Medicine Indiana University School of Medicine Indianapolis Indiana USA

2. Molecular Genetics and Biology of Complex Diseases and Department of Clinical and Molecular Hepatology Institute of Medical Research University of Buenos Aires‐National Scientific and Technical Research Council Ciudad Autonoma de Buenos Aires Argentina

3. Johns Hopkins Bloomberg School of Public Health Baltimore Maryland USA

Funder

National Institute of Diabetes and Digestive and Kidney Diseases

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Hepatology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/hep.32491

Reference48 articles.

1. Genetic variation in PNPLA3 confers susceptibility to nonalcoholic fatty liver disease

2. A nonsynonymous gene variant in the adiponutrin gene is associated with nonalcoholic fatty liver disease severity