Frequent deletion ofCDKN2Aand recurrent coamplification ofKIT,PDGFRA, andKDRin fibrosarcoma of bone-An array comparative genomic hybridization study-Reference-Cited by-同舟云学术

Frequent deletion ofCDKN2Aand recurrent coamplification ofKIT,PDGFRA, andKDRin fibrosarcoma of bone-An array comparative genomic hybridization study

Published:2009-10-27 Issue:2 Volume:49 Page:132-143
ISSN:1045-2257
Container-title:Genes, Chromosomes and Cancer
language:en
Short-container-title:Genes Chromosom. Cancer

Author:

Niini Tarja,López-Guerrero José Antonio,Ninomiya Shinsuke,Guled Mohamed,Hattinger Claudia Maria,Michelacci Francesca,Böhling Tom,Llombart-Bosch Antonio,Picci Piero,Serra Massimo,Knuutila Sakari

Funder

European Commission

Spanish Group for Research on Sarcomas

Publisher

Wiley

Subject

Cancer Research,Genetics

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/gcc.20727/fullpdf

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3. Activating mutations in c-KIT and PDGFRalpha are exclusively found in gastrointestinal stromal tumors and not in other tumors overexpressing these imatinib mesylate target genes;Burger;Cancer Biol Ther,2005

4. Bone and Soft Tissue Tumors

5. KIT mutations are common in incidental gastrointestinal stromal tumors one centimeter or less in size;Corless;Am J Pathol,2002

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