Review: Novel Insights Into Tumor Necrosis Factor Receptor, Death Receptor 3, and Progranulin Pathways in Arthritis and Bone Remodeling
Author:
Affiliation:
1. Cardiff University; Cardiff UK
2. Saarland University Medical School; Homburg, Saar Germany
3. New York University Medical Center; New York New York
Funder
NIH (US Department of Health and Human Services)
Publisher
Wiley
Subject
Immunology,Rheumatology,Immunology and Allergy
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/art.39816/fullpdf
Reference88 articles.
1. Interactions of tumor necrosis factor (TNF) and TNF receptor family members in the mouse and human;Bossen;J Biol Chem,2006
2. Selective inhibition of TFNR1 reduces osteoclast numbers as is differentiated from anti-TNF in a LPS-driven model of inflammatory bone loss;Espirito Santo;Biochem Biophys Res Commun,2015
3. Expression of tumor necrosis factor alpha and its receptors during cellular differentiation;Schling;Cytokine,2013
4. Progranulin is associated with disease activity in patients with rheumatoid arthritis;Cerezo;Mediators Inflamm,2015
5. Progranulin antibodies entertain a proinflammatory environment in a subgroup of patients with psoriatic arthritis;Thurner;Arthritis Res Ther,2013
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