Targeting Protein Kinase CK1δ with Riluzole: Could It Be One of the Possible Missing Bricks to Interpret Its Effect in the Treatment of ALS from a Molecular Point of View?-Reference-Cited by-同舟云学术

Targeting Protein Kinase CK1δ with Riluzole: Could It Be One of the Possible Missing Bricks to Interpret Its Effect in the Treatment of ALS from a Molecular Point of View?

Published:2018-11-21 Issue:24 Volume:13 Page:2601-2605
ISSN:1860-7179
Container-title:ChemMedChem
language:en
Short-container-title:ChemMedChem

Author:

Bissaro Maicol¹,Federico Stephanie²,Salmaso Veronica¹,Sturlese Mattia¹,Spalluto Giampiero²,Moro Stefano¹^ORCID

Affiliation:

1. Molecular Modeling Section (MMS), Department of Pharmaceutical and Pharmacological Sciences; University of Padova; Via Marzolo 5 35131 Padova Italy

2. Department of Chemical and Pharmaceutical Sciences; University of Trieste; Via Licio Giorgeri 1 34127 Trieste Italy

Publisher

Wiley

Subject

Organic Chemistry,General Pharmacology, Toxicology and Pharmaceutics,Molecular Medicine,Drug Discovery,Biochemistry,Pharmacology

Link

http://onlinelibrary.wiley.com/wol1/doi/10.1002/cmdc.201800632/fullpdf

Reference29 articles.

1. TDP-43 Mutations in Familial and Sporadic Amyotrophic Lateral Sclerosis

2. TDP-43 self-interaction is modulated by redox-active compounds Auranofin, Chelerythrine and Riluzole

3. Amyotrophic Lateral Sclerosis

4. Ubiquitinated TDP-43 in Frontotemporal Lobar Degeneration and Amyotrophic Lateral Sclerosis

5. Gains or losses: molecular mechanisms of TDP43-mediated neurodegeneration

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