Socs 3 modulates the activity of the transcription factor Stat3 in mammary tissue and controls alveolar homeostasis
Author:
Publisher
Wiley
Subject
Developmental Biology
Reference27 articles.
1. Suppression of epithelial apoptosis and delayed mammary gland involution in mice with a conditional knockout of Stat3
2. Inactivation of Stat5 in Mouse Mammary Epithelium during Pregnancy Reveals Distinct Functions in Cell Proliferation, Survival, and Differentiation
3. Cytokine-Inducible SH2-Containing Protein Suppresses PRL Signaling by Binding the PRL Receptor
4. Prolactin, Growth Hormone, and Epidermal Growth Factor Activate Stat5 in Different Compartments of Mammary Tissue and Exert Different and Overlapping Developmental Effects
5. Socs2 and Elf5 Mediate Prolactin-Induced Mammary Gland Development
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3. Upregulation of miR-196b-5p attenuates BCG uptake via targeting SOCS3 and activating STAT3 in macrophages from patients with long-term cigarette smoking-related active pulmonary tuberculosis;Journal of Translational Medicine;2018-10-16
4. In vitro significance of SOCS-3 and SOCS-4 and potential mechanistic links to wound healing;Scientific Reports;2017-07-27
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