Subcellular diversion of cholesterol by gain‐ and loss‐of‐function mutations in PMP22-Reference-Cited by-同舟云学术

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Subcellular diversion of cholesterol by gain‐ and loss‐of‐function mutations in PMP22

Published:2020-06-08 Issue: Volume: Page:
ISSN:0894-1491
Container-title:Glia
language:en
Short-container-title:Glia

Author:

Zhou Ye¹,Borchelt David¹,Bauson Jodi C.¹,Fazio Sergio²,Miles Joshua R.²,Tavori Hagai²,Notterpek Lucia¹³^ORCID

Affiliation:

1. Department of NeuroscienceCollege of Medicine, McKnight Brain Institute, University of Florida Gainesville Florida USA

2. Department of MedicineKnight Cardiovascular Institute, Center for Preventive Cardiology, Oregon Health and Science University Oregon Portland USA

3. Department of NeurologyCollege of Medicine, McKnight Brain Institute, University of Florida Gainesville Florida USA

Funder

American Heart Association

Center for Scientific Review

Publisher

Wiley

Subject

Cellular and Molecular Neuroscience,Neurology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/glia.23840

Reference80 articles.

1. Production and characterization of monoclonal antibodies to the extracellular domain of P0

2. Peripheral Myelin Protein 22 Is in Complex with 6beta4 Integrin, and Its Absence Alters the Schwann Cell Basal Lamina

3. Developmental abnormalities in the nerves of peripheral myelin protein 22-deficient mice

4. Dietary cholesterol promotes repair of demyelinated lesions in the adult brain

5. Lipid Metabolism in Peripheral Nerve Cell Culture (Rich in Schwann Cells) from Normal and Trembler Mice

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1. Quantitative proteomics unveils known and previously unrecognized alterations in neuropathic nerves;Journal of Neurochemistry;2024-07-29

2. Roles for PMP22 in Schwann cell cholesterol homeostasis in health and disease;Biochemical Society Transactions;2024-07-09

3. PMP22 duplication dysregulates lipid homeostasis and plasma membrane organization in developing human Schwann cells;Brain;2024-05-14

4. PMP22 associates with MPZ via their transmembrane domains and disrupting this interaction causes a loss-of-function phenotype similar to hereditary neuropathy associated with liability to pressure palsies (HNPP);2023-12-24

5. Therapeutic Strategies for Lysosomal Storage Diseases by Targeting Glial Cells;Physiology and Function of Glial Cells in Health and Disease;2023-12-18

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