Gender specific association ofTP53polymorphisms (EX4 215G>C Arg72Pro, IVS3+40-41ins16, and IVS6+62G>A), with risk of oral cancer subtypes and overall survival of the patients
Author:
Affiliation:
1. Indo-American Cancer Research Foundation; Formerly Research and Development, Basavatarakam Indo-American Cancer Hospital and Research Institute; Hyderabad Telangana India
2. Acharya Nagarjuna University; Andhra Pradesh India
Funder
Indian Council of Medical Research
Publisher
Wiley
Subject
Cancer Research,Molecular Biology
Link
http://onlinelibrary.wiley.com/wol1/doi/10.1002/mc.22543/fullpdf
Reference55 articles.
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4. How many mutant p53 molecules are needed to inactivate a tetramer;Chan;Mol Cell Biol,2004
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1. Analysis of Risk Factors of Oral Cancer and Periodontitis from a Sex- and Gender-Related Perspective: Gender Dentistry;Applied Sciences;2022-09-12
2. Epidemiological, clinical and oncological outcomes of young patients with laryngeal cancer: a systematic review;European Archives of Oto-Rhino-Laryngology;2022-06-02
3. Impact of TP53 polymorphisms (IVS3 + 40-41ins16, EX4 215G > C and IVS6 + 62G > A) on the risk of endometrial cancer, survival and hazard ratio of the patients;Meta Gene;2020-06
4. Meta-Analysis Results on the Association Between TP53 Codon 72 Polymorphism With the Susceptibility to Oral Cancer;Frontiers in Physiology;2018-08-02
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