The T99K variant of glycosylasparaginase shows a new structural mechanism of the genetic disease aspartylglucosaminuria
Author:
Affiliation:
1. Department of Biological SciencesUniversity of Massachusetts Lowell Lowell Massachusetts, 01854
Funder
National Institutes of Health
Publisher
Wiley
Subject
Molecular Biology,Biochemistry
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/pro.3607
Reference39 articles.
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3. Aspartylglycosaminuria: a review
4. Aspartylglycosaminuria (agu). Further aspects on its clinical picture, mode of inheritance and epidemiology based on a series of 57 patients;Autio S;Ann Clin Res,1973
5. Aspartylglycosaminuria: biochemistry and molecular biology
Cited by 2 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Aspartylglucosaminuria: Clinical Presentation and Potential Therapies;Journal of Child Neurology;2021-01-13
2. Disorders of glycoprotein degradation: sialidosis, fucosidosis, α-mannosidosis, β-mannosidosis, and aspartylglycosaminuria;Rosenberg's Molecular and Genetic Basis of Neurological and Psychiatric Disease;2020
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