Reduced glutamate decarboxylase activity in the subthalamic nucleus in patients with tardive dyskinesia-Reference-Cited by-同舟云学术

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Reduced glutamate decarboxylase activity in the subthalamic nucleus in patients with tardive dyskinesia

Published:1989 Issue:1 Volume:4 Page:37-46
ISSN:0885-3185
Container-title:Movement Disorders
language:en
Short-container-title:Mov. Disord.

Author:

Andersson Ulf,Hägström Jan-Erik,Levin Edward D.,Bondesson Ulf,Valverius Milan,Gunne Lars M.

Publisher

Wiley

Subject

Neurology (clinical),Neurology

Reference33 articles.

1. Tardive dyskinesia. Prevalence and risk factors, 1959 to 1979;Kane;Arch Gen Psychiatry,1982

2. The pharmacology of tardive dyskinesia;Klawans;Am J Psychiatry,1973

3. Neurobiological substrates of tardive dyskinesia: the GABA hypothesis;Fibiger;TINS,1984

4. Is tardive dyskinesia due to dopamine hypersensitivity?;Casey;Clin Neuropharmacol,1986

5. Further anomalies in the dopamine receptor supersensitivity hypothesis of tardive dyskinesia;Waddington;TINS,1986

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1. The role of glutamate receptors and their interactions with dopamine and other neurotransmitters in the development of tardive dyskinesia: preclinical and clinical results;Behavioural Pharmacology;2020-05-25

2. Gabapentin reduces haloperidol-induced vacuous chewing movements in mice;Pharmacology Biochemistry and Behavior;2018-03

3. Drug-induced Movement Disorders;Neurologic Clinics;2015-02

4. Chorea and Other Hyperkinetic Disorders;Brain Mapping;2015

5. The tardive syndromes;Principles and Practice of Movement Disorders;2011

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