Phosphatidylinositol 3‐Kinase δ Deficiency Protects From Antimyeloperoxidase Vasculitis
Author:
Affiliation:
1. School of Immunology and Microbial Sciences, King's College London, Guy's Hospital London UK
Funder
Medical Research Council Canada
Kidney Research UK
Guy's and St Thomas' Charity
King's College London
Publisher
Wiley
Subject
Immunology,Rheumatology,Immunology and Allergy
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1002/art.42298
Reference16 articles.
1. Phosphoinositol 3-kinase-γ mediates antineutrophil cytoplasmic autoantibody-induced glomerulonephritis
2. Impaired B and T Cell Antigen Receptor Signaling in p110δ PI 3-Kinase Mutant Mice
3. The PI3K isoforms p110α and p110δ are essential for pre‐B cell receptor signaling and B cell development;Ramadani F;Sci Signal,2012
4. PI3K p110δ regulates T-cell cytokine production during primary and secondary immune responses in mice and humans
5. Phosphoinositide 3-Kinase p110δ Regulates Natural Antibody Production, Marginal Zone and B-1 B Cell Function, and Autoantibody Responses
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1. Update on antineutrophil cytoplasmic autoantibody vasculitis in children;Current Opinion in Rheumatology;2024-07-11
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