Abnormal platelet amyloid‐β precursor protein metabolism in SAMP8 mice: Evidence for peripheral marker in Alzheimer's disease-Reference-Cited by-同舟云学术

Abnormal platelet amyloid‐β precursor protein metabolism in SAMP8 mice: Evidence for peripheral marker in Alzheimer's disease

Published:2019-06-10 Issue:12 Volume:234 Page:23528-23536
ISSN:0021-9541
Container-title:Journal of Cellular Physiology
language:en
Short-container-title:J Cell Physiol

Author:

Chen Lizhi¹²^ORCID,Xu Shicheng³,Wu Tong¹⁴,Shao Yijia¹⁴,Luo Li⁵,Zhou Lingqi¹⁴,Ou Shanshan¹⁴,Tang Hai⁶,Huang Wenhua²,Guo Kaihua¹⁴,Xu Jie¹⁴

Affiliation:

1. Department of Anatomy and Neurobiology, Zhongshan School of Medicine Sun Yat‐Sen University Guangzhou China

2. Department of Anatomy, Guangdong Provincial Key Laboratory of Construction and Detection in Tissue Engineering Southern Medical University Guangzhou China

3. Department of Ultrasound The Third Affiliated Hospital of Sun Yat‐sen University Guangzhou China

4. Guangdong Province Key Laboratory of Brain Function and Disease, Zhongshan School of Medicine Sun Yat‐sen University Guangzhou China

5. Department of Anatomy, School of Basic Medicine Guangdong Pharmaceutical University Guangzhou China

6. Department of Anatomy Guangdong Jiangmen Chinese Traditional Medicine College Jiangmen China

Funder

Natural Science Foundation of Guangdong Province

National Natural Science Foundation of China

China Postdoctoral Science Foundation

Publisher

Wiley

Subject

Cell Biology,Clinical Biochemistry,Physiology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1002/jcp.28921

Reference29 articles.

1. α-Secretase ADAM10 as Well as αAPPs Is Reduced in Platelets and CSF of Alzheimer Disease Patients

2. Secretases as targets for the treatment of Alzheimer's disease: the prospects

3. Aging Selectively Modulates Vitamin C Transporter Expression Patterns in the Kidney

4. Biomarkers for Alzheimer's disease: a controversial topic

5. Lifelong Management of Amyloid-Beta Metabolism to Prevent Alzheimer's Disease

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