Activin A stimulates I?B-?/NF?B and rank expression for osteoclast differentiation, but not AKT survival pathway in osteoclast precursors
Author:
Publisher
Wiley
Subject
Cell Biology,Molecular Biology,Biochemistry
Reference40 articles.
1. Commitment and Differentiation of Osteoclast Precursor Cells by the Sequential Expression of C-Fms and Receptor Activator of Nuclear Factor κb (Rank) Receptors
2. Novel activin receptors: Distinct genes and alternative mRNA splicing generate a repertoire of serine/threonine kinase receptors
3. Protein Kinase B (c-Akt), Phosphatidylinositol 3-Kinase, and STAT5 Are Activated by Erythropoietin (EPO) in HCD57 Erythroid Cells But Are Constitutively Active in an EPO-Independent, Apoptosis-Resistant Subclone (HCD57-SREI Cells)
4. Interleukin-3-Induced Phosphorylation of BAD Through the Protein Kinase Akt
5. RANK is essential for osteoclast and lymph node development
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